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Abstract
Pinus monticola (Dougl. ex D. Don.) maintains a complex defence system that detects white pine blister rust pathogen (Cronartium ribicola J.C.Fisch.) and activates resistance responses. A thorough understanding of how it functions at the molecular level would provide us new strategies for creating forest trees with durable disease resistance. Our research focuses on molecular dissection of P. monticola major gene (Cr2) resistance and quantitative partial resistance. To characterize the Cr2 gene, resistance gene family encoding proteins with nucleotide-binding-site and leucine- rich- repeat (NBS-LRR) was identified and used to search for DNA polymorphisms by genotyping with a modified approach of amplified fragment length polymorphism (AFLP).
Parent Publication
Keywords
high elevation five-needle pines,
threats,
whitebark,
Pinus albicaulis,
limber,
Pinus flexilis,
southwestern white,
Pinus strobiformis,
foxtail,
Pinus balfouriana,
Great Basin bristlecone,
Pinus longaeva,
Rocky Mountain bristlecone pine,
Pinus aristata
Citation
Liu, Jun-Jun; Sniezko, Richard. 2011. Molecular dissection of white pine genetic resistance to Cronartium ribicola. In: Keane, Robert E.; Tomback, Diana F.; Murray, Michael P.; Smith, Cyndi M., eds. The future of high-elevation, five-needle white pines in Western North America: Proceedings of the High Five Symposium. 28-30 June 2010; Missoula, MT. Proceedings RMRS-P-63. Fort Collins, CO: U.S. Department of Agriculture, Forest Service, Rocky Mountain Research Station. p. 272.
